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Notwithstanding recent studies establishing a functional intersection of RNA processing with DNA repair, very few RNA processing factors have been examined for their association with the DNA damage response.
Illuminating the role of the microtubule-associated protein tau in neurodegenerative diseases is of increasing importance, supported by recent studies establishing novel functions of tau in synaptic signalling and cytoskeletal organization.
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As recent studies establish, the paucity of sex-disaggregated ICT data, particularly from developing countries, makes it difficult, if not impossible, to make the case to policymakers for their consideration of gender-related issues in ICT policies, plans and strategies (United Nations, 2014).
Recent studies establish a novel role for PPAR-γ signaling as an endogenous mechanism for regulating transforming growth factor-ß (TGF-ß -dependent fibrogenesis.
Recent studies established that BbCRASP-2 is preferentially expressed in mammals, and elicits robust antibody response in infected hosts, including humans.
Several recent studies establish a role for miRNA in vascular development and angiogenesis [ 3].
Recent studies establish that central and peripheral inflammation occurs in the prodromal stage of the disease and sustains disease progression.
The recent studies established that also Mlh1-Pms1 endonuclease is required for MMR in Exo1-independent MMR subpathway (Smith et al. 2013; Goellner et al. 2014).
Interestingly, a number of recent studies established a functional relationship between the inhibition by S-nitrosylation of PDI, defects in regulation of protein folding within the ER, and neurodegeneration.
Recent studies established that GAPD-1 is not simply a classical metabolic protein involved in glycolytic energy production, but rather a multifunctional protein with specific functions in numerous processes [ 3, 4].
More recent studies established that loss of TREX1 in human cells results in a form of inherited systemic lupus erythematosus (SLE) called Aicardi-Goutières syndrome (AGS) (in collaboration with Dr Yanick Crow) [92,104 106].
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