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Recent reports provided compelling evidence that the bri1-5 and bri1-9 variants of the brassinosteroid receptor BRI1, carrying substitutions in their ECD domains, are retained in the ER and degraded by the ERAD system [63], [72], [73].
In addition, recent reports provided detailed anatomy and physiology analysis of mouse placenta [18] [20], a key organ in PAM pathogenesis and pointed out considerable sharing of cell and molecular features with the human placenta, including the hemochorial barrier and the maternal antibody transmission to the fetus across the placenta [20] [23].
Recent reports provided evidence that CXCL12, through its interaction with CXCR4, could induce directed migration of HeLa cells and hepatocellular carcinoma cells and play a role in cell growth, survival, and scatter [13], [18]– [18].
Recent reports provided the first support for the view that the lkt operon was vertically inherited from the last common ancestor of genus Mannheimia to any ancestor of the diverging species, while changes such as chromosomal rearrangements, pseudogene formation, and deletions of various sizes have occurred in the region upstream of the lkt genes in individual subclades/species [ 20, 21].
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Two recent reports provide new physical information on how the XPA protein recruits the ERCC1 XPF heterodimer to the site of damage during the process of mammalian nucleotide excision repair (NER).
Several recent reports provide support for the hypothesis that growth factor and chemokine-mediated chemotaxis of mast cells within tissues can be an important mechanism for a rapid increase in the number of mast cells at sites of inflammation [1], [5].
Two recent reports provide such a link.
However, recent reports provide abundant evidence challenging this paradigm (reviewed in [ 2, 3]).
Several recent reports provide some comparisons from other settings (Table 2).
Two recent reports provide compelling insights into the role for RANK and its ligand, RANKL, in progestin-dependent mammary tumorigenesis.
Recent reports provide evidence for its critical role in host-mediated tumor progression and angiogenesis, particularly in tumor endothelium [ 49].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com