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Two recent reports provide new physical information on how the XPA protein recruits the ERCC1 XPF heterodimer to the site of damage during the process of mammalian nucleotide excision repair (NER).
Several recent reports provide support for the hypothesis that growth factor and chemokine-mediated chemotaxis of mast cells within tissues can be an important mechanism for a rapid increase in the number of mast cells at sites of inflammation [1], [5].
Two recent reports provide such a link.
Several recent reports provide some comparisons from other settings (Table 2).
However, recent reports provide abundant evidence challenging this paradigm (reviewed in [ 2, 3]).
Two recent reports provide compelling insights into the role for RANK and its ligand, RANKL, in progestin-dependent mammary tumorigenesis.
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Recent reports provided compelling evidence that the bri1-5 and bri1-9 variants of the brassinosteroid receptor BRI1, carrying substitutions in their ECD domains, are retained in the ER and degraded by the ERAD system [63], [72], [73].
In addition, recent reports provided detailed anatomy and physiology analysis of mouse placenta [18] [20], a key organ in PAM pathogenesis and pointed out considerable sharing of cell and molecular features with the human placenta, including the hemochorial barrier and the maternal antibody transmission to the fetus across the placenta [20] [23].
Interestingly, during this literature search we found three recent reports providing experimental demonstration of diazotrophy motivated by an initial genomic identification of putative nitrogen fixation genes [ 16- 18].
However, recent reports providing structural insight into the Nck1 and Nck2 SH2 domains indicate that both binding modules are more or less indistinguishable with respect to their binding specificities and both recognize the consensus motif pYDxV AYST x DEC) [ 22].
Recent reports provided evidence that CXCL12, through its interaction with CXCR4, could induce directed migration of HeLa cells and hepatocellular carcinoma cells and play a role in cell growth, survival, and scatter [13], [18]– [18].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com