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One possible reason for discrepancies between our study and results in the overexpression system might be that PBDEs act via disturbance of recruitment of THR cofactors.
Previous studies have suggested that publication bias is one reason for discrepancies between the results of large RCTs and meta-analyses of small RCTs [ 14- 16].
Several reasons can explain these divergences, and these are discussed here by Z. Gu et al. One possible reason for discrepancies in ω-3 PUFA studies, observed also among the interventional trials performed in cardiovascular patients, is highlighted by B. B. Albert et al. in this special issue.
A principal reason for discrepancies with previous results may be that microscopy, electronic and computing equipment have developed considerably in the past 20 years, and thus allow for much finer delineation of spike heights than the original 1987 study [ 11], particularly among F1 hybrids.
Another potential reason for discrepancies in results of studies to date on diet and PPAR-γ Pro12Ala genotype in relation to body composition is that polymorphisms in the PPAR-γ gene other than the Pro12Ala polymorphism could influence body composition and be in linkage disequilibrium with the Pro12Ala polymorphism.
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Another reason for discrepancy may be related to some problem in translation or comprehension.
The reason for discrepancy between these findings and our study is not clear.
The reason for discrepancy of the results obtained in these studies is presently unclear.
The effectiveness data are compared to available correlations and reasons for discrepancies are discussed.
(Additional reasons for discrepancies are discussed later).
There are several possible reasons for discrepancies between studies.
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