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Lipid peroxidation was measured by thiobarbituric acid reactive substances.
ETT-depleted cells displayed enhanced lipid peroxidation monitored by the thiobarbituric acid reactive substances (TBARS) assay (Figure 3b).
Production of thiobarbituric reactive substances (TBARS) in ruminal fluid was reduced by dietary FM.
Estradiol significantly reduced a hypoxia-induced increase in brain thiobarbituric acid reactive substances (P<0.05).
Serum thiobarbituric acid reactive substances (TBARS) were estimated to assess oxidative stress.
Color, thiobarbituric acid reactive substances (TBARS), olfactory and sensory characteristics were evaluated.
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Protein carbonyls and thiobarbituric acid-reactive substances were determined in perfused heart homogenates as described [59, 60].
Moreover, a significant decrease of thiobarbituric acid-reactive substances (TBARs) was observed.
Lipid peroxidation was measured by the production of thiobarbituric acid-reactive substances in a cell-free lipid system.
The liposome oxidation was monitored by determining thiobarbituric acid-reactive substances (TBARS).
As an index of lipoperoxidation, thiobarbituric acid-reactive substances (TBARS) were assayed as previously described [21].
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