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These synthetic elements were introduced in a Drosophila reactive strain, and SINE retroposition was assessed following dysgenic crosses that are known to induce high levels of I factor germinal transposition.
No I-5' or I-m transcripts were found in the JA strain, as expected from a reactive strain mainly containing defective copies that have lost most of their 5' and internal portion.
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According to Peri [21], reactive strained siloxane sights facilitate the chemisorption of NH3 resulting in Si-NH2 groups on silica surface.
By contrast, reactive strains only contain the defective heterochromatic copies and lack functional elements [1], [3].
Lewis and WKY rats were the less reactive strains.
It has thus been possible to study how immunologically cross-reactive strains sequentially invade a partially susceptible population.
Two main modelling approaches have been used to study immunologically cross-reactive strains: (i) history based (HB) models [4] and (ii) status based (SB) models [20].
These sublines were therefore analyzed by semi-quantitative RT-PCR, using for control the JA reactive (R) strain, which contains only defective, retrotranscriptionally inactive I elements located in pericentric heterochromatin.
I factors of Drosophila melanogaster are LINE-like elements that actively transpose in the germline of the female progeny (called SF) from crosses between females of reactive (R) strains and males of inducer (I) strains [1]; for a recent review see [2].
To determine whether B. pertussis vaccines induce antibodies that recognize and/or protect against B. parapertussis, we first tested whether antibody responses are cross-reactive between strains and if O-antigen affects the responsiveness of antibodies.
Anti-HRSV antibody directed against F protein is cross-reactive for strains of both subtypes [1], [2], [28], [29], and studies on HMPV using human sera and animal models have indicated similar antibody reactivity patterns [30] [33].
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