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Serum leptin and total non-esterified fatty acids (NEFA) were elevated in HFD rats, which also had reduced bone mass compared to LFD-fed animals.
To investigate the impact of IL-6 on mortality after trauma plus hemorrhage, SBR35 rats, which also experienced HCC and cardiomyocyte apoptosis (Table 1 and Figure 2), were randomized to receive either placebo or IL-6 (10 µg/kg) at the start of resuscitation (Figure 5).
MIF-like staining occurred in both CYP-treated and saline-treated rats, which also showed a substantial variation within each groups.
The level of MDA was reduced in both AM- and Silymarin-treated rats which also suggested the protective and curative activities of AM against liver damages.
Our results correspond with those found by Jowsey [ 72], as the femurs were longer in the control rats, which also displayed higher values for osteons.
As mentioned above, we have observed a reduction in ACE2 activity in the medulla in STNx rats, which also had polyuria.
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Additionally, this vasodilation was diminished in their diabetic littermates, the Zucker diabetic fatty (ZDF) rat, which also exhibited endothelial dysfunction.
Alignment of mouse and rat genomic sequences resulted in information for rat exons, which also consist of 11 exons.
The benign properties of the sponges were demonstrated in an in vivo subcutaneous rat model, which also revealed uniform infiltration of vascularized tissue by 8 weeks and complete degradation of the sponges by 16 weeks, with only a minimal inflammatory response being observed over the course of the experiments.
This also demonstrates a conservation of outcomes between human cells and rat cells, which also did not require prolonged survival to provide durable benefit [14], [57], suggesting that this too might be a conserved aspect of GDABMP function.
This is surprising, given that the residues in rat MST3 {MST3(274–306), which also contain the bipartite NLS [ 28]} are homologous to YSK1/SOK1(24/3302) (24/33 identity with 4/33 conservative replacements).
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