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Rats were identified as high-responder (HR; 15% most active) or low-responder (LR; 15% least active) rats based on the locomotor response.
All rats were identified using a microchip (identity chip, Animal Care Ltd ,York, UK).
Six neurons (n = 6 rats) were identified as striato-nigral MSNs by antidromic activation of the SNr (Fig. 8A).
Rats were identified by ear punch in accordance with the Jackson Laboratory system.
Oestrous cycle phases of intact WKY rats were identified by vaginal smear.
The rats were identified using mitochondrial DNA sequence analysis and karotyping (J. Patton, University of California, Berkeley, CA, pers. comm).
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Lower expression of klotho in medulla oblongata of diabetic rats was identified.
The infection agent both in blue foxes and rats was identified as T. spiralis.
Work from our lab has reported that nestin downregulation in the heart of type I diabetic rats was identified as an incipient pathophysiological event and contributed in part to the impaired neurogenic response of neural progenitor/stem cells during the reparative fibrotic response of the type I diabetic infarcted rat heart [ 15, 16].
Homologues of the genes for mouse and rat were identified using the NCBI's HomoloGene release 64.
Dot matrix view of human EC Kim-1 domain and rat's EC Kim-1 domain and Ig like V subdomain aligned in BLAST program is shown in Figure 2. Rat and human cDNAs encoding KIM-1 (KIM-1 in the rat) were identified for the first time using difference analysis between normal kidneys and kidneys exposed to ischemia/reperfusion (I/R) injury followed by regeneration of proximal tubular cells.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com