Using a CSD rat model, we investigated if valproate and topiramate lower the susceptibility to develop CSD by inducing changes in brain DNA methylation.
Using a rat model, we previously showed that chronic administration of lipopolysaccharide and proteases to gingival sulcus induced both periodontal inflammation and liver injury.
Using cultured alveolar interstitial fibroblasts and an in vivo perinatal nicotine exposure rat model, we found that PPARγ promoter methylation is strongly correlated with inhibition of PPARγ expression in the presence of nicotine.
Using a rat model, we tested a new bioengineering strategy for the in vivo and de novo generation of autologous grafts without the addition of extracellular matrix or cells, and analyzed their biomechanical and structural properties.
Hypermetabolic patterns in PD patients are often interpreted as compensatory changes [31] and so, assuming this to be true for our rat model, we would expect strong correlations with compensatory gait changes (increased IH print area and duration of CH, IF and IH stance).
In the 6-hydroxydopamine (6-OHDA) PD rat model we investigated the effect of STN DBS on deficient prepulse inhibition (PPI) induced by the dopamine (DA) receptor agonist apomorphine, which is an operative measure for disturbed sensorimotor gating seen in certain neuropsychiatric disturbances.
Using our rat model, we investigated the effect of the yeast alternative NADH dehydrogenase, Ndi1.
In order to further characterize the Fischer 344 rat model, we determined how the various F. tularensis strains replicated in rat BMDM.
Based on recent findings with a Wistar rat model, we speculated that the γ-secretase pathway may be associated with atherosclerosis.
Using the rotenone rat model, we investigated the protective effects of alternative NADH dehydrogenase (Ndi1) which we previously demonstrated to act as a replacement for complex I both in vitro and in vivo.
In the present study, by using the same acute peritonitis rat model, we explored the effect of the L. saccharina-derived fractions L.s.-1.0 and L.s.-1.25 on PMN transmigration.
