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From our results, it is noted that PD was not related to lack of response of the target tumor but rather to the development of new lesions.
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It is claimed that most of the variance in such data are due to cohort differences rather than to the development of memory.
American and German physician organizations, including the American Medical Association (AMA) and the German Medical Association (BÄK), have been rather opposed to the development of PRSs, arguing, for example, that the identity of patients cannot be confirmed, reviews will be excessively negative, and physicians' responses will be hampered by confidentiality issues [ 16].
Due to known limitations of either pure content-based recommender systems or collaborative filtering, it rather soon led to the development of hybrid recommendation that combine the advantages of different recommendation techniques.
This suggests that variations in retinal vascular D f are likely consequential, rather than antecedent to, the development of diabetic retinopathy.
Quantitative data on Z. subfasciatus egg laying and adult emergence across all the genotypes showed that these two traits were not correlated, therefore the mechanism of resistance was not based on any antibiosis at the seed coat level but rather on antibiosis to the development of the insect within the seed as was known previously.
Furthermore, the data clearly show that the decreased barrier integrity observed in DKO relative to Il10−/− mice occurs prior to, rather than in response to, the development of colon inflammation.
Our interest in the immediate aftermath of trauma is to understand preventative (rather than just curative) measures to the development of PTSD flashbacks.
Rather, they are usually due to the development of an ignored fistulae that descends to the mediastinal space.
38 Hence, it is likely that in multiply infected women some HPV types give rise to different lesions and some HPV types are not clinically relevant, rather than multiple types contributing to the development of a single lesion.
Assuming that sema3A and NP-1 are essential for the regulatory function of B cells, one would expect to see that when their expression on B cells is decreased, B cells may shift, to become more pro-inflammatory rather that regulatory, and contribute to the development of SLE.
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