Sentence examples for rate of cholesterol from inspiring English sources

The highest rate of cholesterol synthesis by the body occurs in the liver, although cholesterol is also made in the intestines, adrenal glands, CNS, and reproductive organs.

In fact, the rate of cholesterol synthesis in the CNS exceeds the need for new cholesterol, so that some cholesterol must move out of the CNS through excretory pathways.

Experimental data demonstrate that the initial extraction rate of cholesterol increased with temperature following static ethanol-modified SC-CO2 extractions at 300 bar.

The rate of cholesterol movement from the plasma membrane to the endoplasmic reticulum is higher in cells with increased cholesterol levels, leading to an enhancement of the storage process in cells with excess unesterified cholesterol (Wustner et al., 2005).

Disruption of the murine cholesterol 24-hydroxylase gene reduced the rate of cholesterol synthesis in the brain by 40%, whereas the brain content of cholesterol was unaltered (Lund et al., 2003), this is possibly due to the concomitant reduction in the cholesterol mevalonate pathway.

The rate of transfer of 7DHC by N-62 StAR protein was comparable to rate of cholesterol transfer by this carrier.

Supersaturation of bile with cholesterol precedes the formation of cholesterol gallstones and this can be caused by a low rate of cholesterol-7α-hydroxylation [ 10].

Aim of this study was to correlate the levels of this metabolite with the rates of cholesterol 7α-hydroxylation in vivo, a direct measurement of bile acid synthesis, in hyperlipidemic patients.

Although all the patients in the analysis had Medicare coverage, blacks were significantly less likely than white women to have seen a primary care doctor in the 6 to 18 months before diagnosis, and they had far lower rates of cholesterol and colon cancer screening.

Statins are a group of medicines that can help lower rates of cholesterol in the blood.

In cells from heterozygotes, a 60percentt reduction in LDL receptors leads to a concentration-dependent defect in regulation, so that attainment of equal rates of cholesterol synthesis and LDL degradation in normal and heterozygous cells requires a two- to threefold higher concentration of LDL in the heterozygote.