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The construction of diabetic rat models is described in Materials and Methods.
In relation to cardiac research, one of the limitations of current rat models is the extended periods taken to develop cardiac phenotypes, despite the presence of abnormal circulating metabolites from an early age.
The generation of these rat models is summarized in Box 2. The Apc Pirc/+ (Pirc) rat was developed by targeted ENU-induced germline mutagenesis (van Boxtel et al., 2010) of the inbred F344/NTac rat strain (Amos-Landgraf et al., 2007).
Type 1 diabetes SD rat models were prepared according to the previous report [4, 10].
The rat models were then allometrically scaled to obtain models for inhalatory exposure for man.
A study using healthy rat models was designed to examine if the nanoparticles had a toxic effect on the model knees [71].
PAH rat models were established by a single intraperitoneal injection of 1% monocrotaline (MCT, 50 mg/Kg).
Type 2 diabetic rat models were induced by means of a high fat diet and intraperitoneal injection with streptozotocin.
Adverse ventilation has in rat models been correlated to edema formation and TNFα production [26, 27], whereas low VT ventilation has been correlated to the opposite picture [28].
Young and aged rat models are relatively homogenous in their gene expressions in the various organs.
From the first day that the rat models were made, different drugs were intragastrically administrated with the specified dosage once a day for 7 days.
