Sentence examples for rat models demonstrated from inspiring English sources

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Imaging with 123I-CMICE-013 of chronic occlusion and ischemia/reperfusion rat models demonstrated excellent images of myocardial defects, which correlated well with ex vivo staining with triphenyltetrazolium chloride (TTC) [6].

Diabetic rat models demonstrated a similar dysregulation in the renal microvasculature early in diabetes with severe insulin deficiency.

It is noteworthy that recent studies on mouse and rat models demonstrated that the methylation alteration with age is highly tissue dependent [ 69, 70].

Studies in mature cortical neuron cultures and rat models demonstrated that approximately 32.4%% of the synaptic spontaneous miniature excitatory postsynaptic currents (mEPSCs) are mediated by NR2B and 26.6 % of the extrasynaptic mEPSCs are mediated by NR2A [ 3].

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Both ADRP rat models demonstrate the activation of the UPR at P21 and P30.

Induction of AKI in ischemia-reperfusion rat models demonstrates decreased OAT1 and OAT3 mRNA as well as protein expression [ 34- 36].

In vivo assessment in a subcutaneous Wistar rat model demonstrated that the DAT bioscaffolds were well tolerated and integrated into the host tissues, supporting angiogenesis and adipogenesis.

Orally dosed pharmacokinetics in a rat model demonstrated a 176% increase in SN38 blood plasma exposure in comparison with a raw drug suspension and a significant increase in the period where therapeutic levels are established.

Recent work in the DR-BB rat model demonstrated a synergistic effect of these two drugs when used together (17).

Studies using a rat model demonstrated that endostatin was upregulated after myocardial infarction and that an antiendostatin antibody promoted mortality [ 19].

The first study using OA-induced hyperuricemic rat model demonstrated that hyperuricemia induced systemic hypertension as well as ischemic type of kidney injury with collagen deposition, macrophage infiltration, and increase in tubular expression of osteopontin documented via immunohistochemical stains.

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