Using the rat model, the present study demonstrates some limitations.
By applying a monocrotaline (MCT -induced rat MCT -inducedffect of Ost on ratremodeling was investheateffect
For the rat model, the S value for 131I was 70%and90%0% lower than that for 123I and 125I, respectively.
For the rat model, the S value was 340% and 230% higher for 131I than that for 123I and 125I, respectively.
For the rat model, the S value for 131I was 6% higher and 50% lower than that for 123I and 125I, respectively.
In a Huntington's disease rat model, the local administration of nerve growth factor (NGF -loaded PLGA eNGF -loadedoPLGAenabledafter excitotoxineuroprotectionid injections [125].
From the Ames test and subacute toxicity test using oral administration in a rat model, the cordycepin was proved to be a non-mutagenic and non-toxic compound.
The objective of this study was to evaluate, in a rat model, the effect of hyperbaric oxygen (HBO) on the healing of normal bone on day 7.
In the RA rat model, the results of swelling inhibition on primary arthritis of bilateral hind paws further confirmed the above-mentioned point.
In a rat model, the superficial inferior epigastric vessels were isolated, ligated, and then threaded through a prefabricated coral cylinder (hydroxyapatite, ProOsteon 500).
In a middle cerebral artery occlusion (MCAO -induced post-ischeMCAO -induced, the BBB (blood brain barrier)-detoured intranasal administration of Ca2+/ATP/bPEI1.8kDa NCs induced a better reduction in infarct volume and neurological deficits than did free ATP.
