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LPPs retention in the rat lung was carried out using bronchoalveolar lavage fluid method.
Three PCRs with Psmb8 in rat lung, Cnot2 in rat liver and Prpf4b in M.fortis lung produced products that were relatively under-expressed compared with the microarray data while a PCR amplification of Abhd6 in rat lung was relatively over-expressed.
The use of rat lung was necessitated by the lack of sufficient proliferation of P. carinii in culture.
When a membrane protein fraction from rat lung was subjected to I-peptide affinity chromatography, the four SR splicing factors were isolated and identified by mass spectrometry.
In addition to exhibiting higher basal glucose uptake, female rat lung was also more responsive than male lung to E2 treatment (Hart et al., 1998).
To isolate AEC II, rat lung was perfused using solution II (0.9% NaCl, 0.1% glucose, 30 mM HEPES, 6 mM KCl, 0.1 mg/ml streptomycin sulfate, 0.07 mg/ml penicillin G, 0.07 mg/ml EGTA, 3 mM Na2HPO4, and 3 mM NaH2PO4, pH 7.4) to clear the red blood cells.
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Transcripts of γ-GCS-LS in rat lung are maximally elevated (8-fold) 2 h following Cd inhalation exposure and remain significantly higher than air controls at 24 h.
In addition to standard vector screens, several genomes of potential microbes living within the immunosuppressed rat lung were added to the screening library.
Interestingly, steady state retinoid levels found in rat lung are rather high compared to other tissues [18].
In contrast, the percentage of nonmuscularized arteries in saline-treated MCT-PH rat lungs was 1.2±0.4%, and the percentages of partially and nonmuscularized arteries were 33.3±4.3% and 65.4±4.5%, respectively.
Another marker that was altered in propranolol-treated rat lungs was the PUFA/MUFA ratio.
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