Sentence examples for rat kidney epithelial from inspiring English sources

Similarly, upregulated ATP1A1 expression is reported in ouabain-treated rat kidney epithelial cells [55], offseting the enzyme inhibition.

To this end, we cloned wild-type and mutant TRKB into the retroviral pBabe-puro expression vector and transduced rat intestinal epithelial (RIE-1) cells as well as E1A-immortalized rat kidney epithelial (RK3E) cells.

Thus, plakoglobin overexpression in transformed rat kidney epithelial cells promotes unregulated growth and foci formation (Kolligs et al, 2000).

More direct evidence was presented by Kolligs et al (2000), who demonstrated that overexpression of wild-type plakoglobin elicits neoplastic transformation in a rat kidney epithelial cell line.

Similar conclusions about CSL-dependent signalling were reached in an analysis of epithelial cell transformation by Notch1 and E1A in rat kidney epithelial cells [ 34].

The rat kidney epithelial cell line, NRK-52E was transiently transfected with vector-only (CT), WT PKD2, R742X PKD2 and 1 702 PKD2 (a PKD2 mutant lacking the entire carboxy-terminal region of the protein).

The siRNA/DAAO was effective in blocking DAAO expression in NRK-52E rat kidney tubule epithelial cells, compared to the nonspecific oligonucleotides.

All the compounds were evaluated for their inhibition on extracellular matrix component (fibronectin) generation by using TGF-β1 induced rat kidney tubular epithelial cells.

Considering that C. spicatus is commonly used for renal diseases, in this study, compounds 1 3 were evaluated for their roles in renal protection by targeting fibronectin production in TGF-β1-induced rat kidney tubular epithelial cells using the method reported previously [13].

HDAC2 is also upregulated in TGF β-induced EMT in rat kidney tubular epithelial cells and streptozotocin-induced diabetic nephropathy.

Overexpression of endogenous regucalcin was found to suppress apoptosis in modeled rat hepatoma cells and normal rat kidney proximal epithelial NRK52 cells induced by various signaling factors.