Sentence examples for rat heart is from inspiring English sources

While in vivo the coronary flow through the rat heart is 3 ml/min, in the buffer-perfused Langendorff heart, this increases to approximately 14 ml/min at the same perfusion pressure, due to the lower oxygen-carrying capacity of Krebs buffer.

The role of IKr and IKs for action potential repolarization in rat heart is still poorly understood [ 32].

Since less than an hour of ischemia-reperfusion in an isolated rat heart is required to demonstrate intracellular redistribution of Cx43 [ 12], we predicted that pacing-induced gap junction remodeling might occur over a short term.

The squid light organ database is dominated by signal transduction (67%) and membrane-associated proteins (49%), whereas the database of the rat heart is dominated by cell growth and maintenance (63%) and intracellular activities (70%).

There are many well-characterized rat models of cardiac disease, and the organization of myocytes and perimysial collagen in the normal rat heart is qualitatively similar to that observed in the dog (27) and pig (13).

Liposomes embedded with membrane proteins prepared from rat heart were allowed to bind to N-terminally immobilized salusin-β (lane 3).

The contraction of isolated rat heart was measured in the presence or absence of PKA inhibitor H89 or propofol, using a pressure transducer.

Isolated perfused rat heart was subjected to global ischaemia for 30 min followed by reperfusion for 120 min. Coronary effluent was analysed for LDH and CK release to assess the degree of cardiac injury.

Isolated perfused rat heart was subjected to global ischemia for 30 min followed by reperfusion for 120 min. Coronary effluent was analyzed for LDH and CK-MB release to assess the degree of cardiac injury.

Isolated perfused rat heart was subjected to global ischaemia for 30 min followed by reperfusion for 120 min. Coronary effluent was analysed for lactate dehydrogenase and creatine kinase enzyme release to assess the degree of cardiac injury.

They suggested that capsaicin-induced coronary artery spasm in the rat heart was mediated by endothelin release from sensory nerve terminals, resulting in deteriorated cardiac function.