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The magnetic hydrogel was injected into a rat brain using stereotactic surgery.
Thus, the present study was designed to determine the detailed localization of 5αR2 in the adult rat brain, using a highly specific polyclonal antibody against this isoform.
We compared the consequences of neonatal exposure (postnatal days PN1-4) to diazinon or parathion on development of norepinephrine systems in rat brain, using treatments designed to produce equivalent effects on cholinesterase, straddling the threshold for barely-detectable inhibition.
This study was designed to determine whether or not kallikrein has a potential to hydrolyze IGFBP-5 and their topographic proximity was investigated in rat brain using double immunohistochemical staining method.
Sagittal sections (8 μm) were prepared from a frozen rat brain using a cryostat (HM560, Carl Zeiss, Oberkochen, Germany).
Contrary to the in vitro result, in vivo specific binding was not observed in the rat brain using PET with [11C]MMPIP (Figure 4).
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The region of rat brain, used for analysis, was determined according to the brain atlas [ 51] from the level 14 (+0.95 from bregma) dorsally in a sequence of 3 slices, each 7 μm thick, in the distance of 15 sections (i.e., approximately 105 μm); it means that analysed part of rat brain was of about 220 μm thick.
To develop an accurate method for quantifying the number of hNSC incorporated in rat brain, we performed real-time PCR on hNSC-transplanted rat brains using a target sequence for ERV-3, which is an endogenous retrovirus present with a known copy number in all human cells, but not present in rodent cells.
This area was first discovered at the caudal border of the striatum and the surrounding areas on the rostral edge of the globus pallidus in rat brains using histochemical techniques in 1988 [1].
The researchers recorded currents running through neurons in the memory and learning centers of rat brains, using a technique unavailable to Hodgkin and Huxley called the patch-clamp method.
Therefore, we have assessed the effects of a therapeutic equivalent dose of olanzapine (2.5 mg/kg per day for 21 days), a commonly used antipsychotic, on DNA methylation in rat brains using rat methylation arrays.
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