Five carcasses from each group were randomly electrically stimulated for 60 s, nearly 20 min postmortem using V1.3-R3B stimulator (7.5 ms duration positive pulses only and an output of 90 95 V, AgResearch, New Zealand).
Our results show that it does not really matter where a culture is stimulated; randomly varying stimulation sites induce comparable connectivity changes as a single selected stimulation electrode.
Axons connected by gap junctions can produce very fast oscillations (VFOs, > 80 Hz) when stimulated randomly at a low rate.
The random network has the same parameters as the random network in Traub et al. (1999), and axons are stimulated randomly via a Poisson process with a rate of 2/s/axon.
Neurons in L0 are stimulated randomly at low frequency by external sources, e.g. at 5 Hz by Poisson spikes.
We conducted simulations in which only half of the granule-cell clusters that were randomly chosen were stimulated by the CS signal while the rest were exposed to the 5-Hz background activity.
Bone morphogenetic protein 2 (BMP2) and type I collagen gene expression were higher on randomly-oriented scaffolds stimulated with TGFβ3, suggesting this substrate was more supportive of an endochondral phenotype.
Protein samples from COPD patients, healthy control or in-vitro stimulated neutrophils were randomly labeled with Cy3 or Cy5.
By contrast, the Golgi were aligned randomly when cells were not stimulated or allowed to undergo BMP2-induced chemotaxis in the presence of PI103.
We stimulated at single electrodes which were randomly chosen before each next stimulus pulse.
Patients were randomly divided into two groups of equal size for being predominantly stimulated either on the left (four patients) or right (four patients) hemisphere.
