This hypothesis is supported by the fact that this RAFT TE was far thicker than any of the other RAFT TEs tested in this study.
To deeply analyze the importance of VEGF/VEGFR-2 interaction to induce ROS production and related glucose uptake [ 21] and the downregulation exerted by VEGFR-2 localization in caveolae/lipid rafts, we tested, in control and serum starved cells, different compounds able to inhibit the binding of VEGF to its receptors.
Bubble trawls significantly enhanced Calanus concentrations in the upper water column: up to 1416%, with the best results for the transverse sparger raft, which also was tested with weak stratification.
A range of different polymers based on styrene, acrylates, methacrylates and acrylamides were synthesised with different RAFT agents and successfully tested for thermolysis at temperatures between 220 and 250 °C in a stainless steel tube flow reactor, resulting in complete removal of thiocarbonylthio end-groups.
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In order to evaluate the role of these structures in elastin peptide-induced signaling, we depleted the cholesterol from rafts using MCD and tested afterwards the ability of elastin peptides to trigger ERK 1/2 activation (Fig. 2).
Having observed reduced CD44 raft affiliation in migrating highly invasive cells, we sought to directly manipulate CD44 raft affiliation to test its impact on cell motility.
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Immune and non-immune mouse sera were tested for anti-RAFT antibodies by using direct ELISA, as we recently described [21], [82].
As APP processing is often located in lipid rafts reliant on cholesterol, we tested statins and cholesterol chelator M βCD.
