Adjusted probabilities of each intervention are calculated for each income quintile, controlling for age and comorbidities.
Similarly, total fluid intake not including water was not associated with risk (OR = 0.84; 95% CI, 0.53 1.33, for highest vs. lowest quintile comparison, controlling for water in the model).
For whole fruit, a slightly lower risk was observed in the 2nd and 4th quintiles as compared with the lowest quintile after controlling for known risk factors of GDM.
Women whose ER- α and ER- β serum bioactivity were in the fifth quintile of controls had a 7.57- 95% CI: 2.46 23.32) and a 10.14 955% CI: 3.19 32.23 -fold risk for general and ER-positive breast cancer, respectively.
Conditional logistic regression models were applied to calculate the relative risks (RRs) (odds ratios) for prostate cancer in relation to fifths of genistein, daidzein, enterolactone and enterodiol concentrations, using cut points defined by the quintiles among control participants for all centres combined and using the lowest category as reference.
In the Nurses Health Studyy [ 5], serum ferritin levels and the sTfR-to-ferritin ratio were associated with an approximately 2.5-fold higher diabetes risk in the highest vs lowest quintile after controlling for C-reactive protein and various conventional risk factors of diabetes.
Conditional logistic regression models were applied to calculate the relative risks (odds ratios) for prostate cancer in relation to fifths of genistein concentration using cut points defined by the quintiles among control participants for all centers combined and using the lowest category as reference.
Women whose levels of serum ER- α and ER- β bioactivity were in the highest quintile among controls had a 7.57- 95% confidence interval (CI): 2.46 23.32; P=0.0004) and a 10.14 955% CI: 3.19 32.23; P<0.0001 -fold risk for general and oestrogen receP<0.0001 -foldbriskt cancer, respectively.
The values for oestradiol, ER- α and ER- β serum bioactivity were classified according to quintiles among controls.
It is evident from the distribution of lifetime average iAs concentration (quintiles of controls) shown in Table 2 that much of the population (first three quintiles) had exposure < 7 μg/L.
