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For this purpose, mouse models of NSCLC are especially useful since the efficacy of new drugs can be tested in lung cancer tumors of specific histology.
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For this purpose, mouse models expressing mutated genes specifically in the lung have been implemented.
For this purpose we used two mouse models of intestinal inflammation - where disease is driven primarily by innate cells or by CD4+ T cells and thus replicate distinct components of human IBD.
The results obtained in mouse models of cancer are promising but any attempt to modulate p38 MAPK activity for therapeutic purposes should be carefully evaluated in preclinical models.
There are now mouse models of Parkinson's.
There are already many mouse models of human diseases in which a simple genetic mutation in the mouse causes symptoms reminiscent of the human disorder.
Research on mouse models of motor neurone disease (MND) have demonstrated the important of a growth factor called VEGF.
Several mouse models of HD are available.
We further demonstrated therapeutic efficacy in mouse models of DMD.
Now, of course, one can easily argue that mice are not people, and that mouse models of Lyme disease do not adequately reproduce what happens in people.
For this purpose, we used a transgenic (TG) mouse model of CD11c-DTR in which CD11c+ cells can be depleted after DTx injection (Jung et al, 2002).
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