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Additional support for the sensitivity of the FTMT to right frontal lobe functioning is provided by the finding of a significant negative correlation between performance on Part D of the FTMT and high beta magnitude.
Additional confirmation of the ability of HSV-1 to alter APP processing was provided by the finding of a 30-kDa N-terminal APP-F in the supernatants from infected but not control cells.
The first evidence of an oscillator coupled to somite segmentation was provided by the finding of the oscillatory expression of the basic helix loop helix (bHLH) gene c-hairy1 in the chick PSM [3].
The rationale for the present study was provided by the finding of obesity in Atxn2 knockout mice [12] and the presence of linkage signals for obesity-related traits on 12q24, the location of the human ATXN2 gene [14], [15], [16].
Additional evidence of a role of SIM1 haploinsufficiency in human obesity was provided by the finding of rare non-synonymous SIM1 mutations enriched in severely obese patients in comparison with lean individuals [ 33].
Evidence of recent duplication activity playing a crucial role also in the evolution of Anopheles female proteases is provided by the finding of an additional copy of AGAP005196 located in the same gene cluster.
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Further evidence confirming which of them is involved in the activation of caspase-3 is provided by the findings of elevated levels of caspase-8 and Fas, coupled with no alteration or even reduced levels of caspase-9 activity and cytosolic cytochrome c in ventricles of rats exposed to SHS.
Evidence of inequity is provided by the finding that residents of districts with a high incidence rate for lung cancer are less likely to receive any treatment after adjusting for deprivation, first hospital attended and case mix.
Further evidence that DNA damage may be a biomarker to predict irinotecan response was also provided by the finding that none of the patients with PD exhibited saturation of the dose response curve at doses lower than 5 μmol/L SN-38, whereas the response plateaued in some of those with clinical benefit.
A strong indication that cell autonomous effects are indeed at the basis of microglia hyper-excitability has been provided by the finding that expression of a mHTT transgene specifically in myelomonocytic cells is sufficient to induce such phenotype [ 35].
Further statistical support was provided by the finding that none of the other significant SNPs improved the R2 value when tested in a stepwise model alongside Phe/Tyr209.
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