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Various environmental risk factors have been proposed to play a role in the etiopathogenesis of schizophrenia.
Several genetic variations have been proposed to play a role in giving African athletes a possible advantage in endurance sports.
Changes in the food environment such as the abundant presence of food cues have been proposed to play a role in the increased prevalence of obesity [1, 2].
Cellular immunity, altered in pregnancy and the peripartum period, has been proposed to play a role in PPCM pathogenesis.
This is proposed to play a role in the deteriorating eye health of patients despite the use of antibiotics.
Recently, hydrogen-related impurities/defects are proposed to play a role in the n-type conductivity in ZnO [33, 48].
Previously, CaMKIV was shown to stably associate with protein serine/threonine phosphatase 2A (PP2A), which was proposed to play a role in negatively regulating the kinase.
In mitosis, Cyclin B1 undergoes phosphorylation at several sites, a subset of which have been proposed to play a role in Cyclin B1 accumulation in the nucleus.
Instead, ESK1 was proposed to play a role in water uptake or circulation [14], [15].
Aberrant expression of BORIS has been proposed to play a role in tumorigenesis [7].
This nuclease has been proposed to play a role during apoptotic chromatin degradation [53], [54].
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