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An administration official said the governor would propose that the changes take effect beginning in 2014; the proposal would not affect this year's race for mayor of New York.
Considering that expression changes occur only after 1h of L-dopa, we propose that the changes actually reflect changes in PTM status instead of altered protein translation.
While the hypotheses to explain it at regional scale are still underway [43], [45], in this work we propose that the changes observed in the Adriatic and Ligurian seas are an ecosystem-wide response to some type of large scale forcing.
Since numerous in vitro studies showed that E-cadherin loss and increased FSP-1 expression are induced by the action of TGFβ [26] [28] and since TGFβ is considered as a major mediator of the pro-fibrotic action of Ang II [29] [31], it is logical to propose that the changes in E-cadherin and FSP-1 expressions in the RenTg mice are due to the Ang II-induced activation of TGFβ pathway.
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We propose that the change of acceleration (i.e., d3 z/d t3 ∝ A(2π f)3) is important for liquefaction because it causes relative motion between the particles.
We propose that the change involved the mechanism reported here, by which additional paralogs were generated by NAHR (Shaffer and Lupski 2000; Stankiewicz and Lupski 2002) in the manner proposed for LCRs (Lupski 1998; Stankiewicz and Lupski 2002).
However, we propose that the change in microparticle surface porosity at pH 7 is an effect of the amelogenin solubility profile and the tendency of amelogenin to self-assemble at neutral pH.
McCarthy et al., [64] proposed that the changes they observed in neurons infected with PRV-Bartha 152 might be responsible for the late neurological abnormalities exhibited by the infected mice.
The authors proposed that the changes in expression of these two genes may reflect a role in homogalacturonan synthesis, which accumulated to a high level in old leaves.
It has been proposed that the changes in the RA synovium are a random process, caused by, for example, altered expression of p53 [ 8, 9].
It is proposed that the changes in hemodynamics, for example, hypotension and atherosclerosis, may play a role in the genesis of microinfarcts in watershed regions.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com