Sentence examples for proportion of pathogenic from inspiring English sources

The pool of bacterial genomes was split into 5 subsets, selecting the organisms randomly within each of the two classes P and N, but making sure that each subset had the same proportion of pathogenic and non-pathogenic bacteria.

The ensuing minor, self-limiting inflammatory response may serve to maintain the mostly beneficial population of intestinal bacteria while providing a sampling of intestinal contents that could lead to an early warning of changes in the proportion of pathogenic bacteria in the intestinal lumen.

A significant proportion of pathogenic BRCA1 and BRCA2 germline mutations comprise large rearrangements in many populations [ 34, 35], and a method suitable for use on FFPE tissue would still be required to detect this class of mutation.

We compare the proportion of pathogenic and benign CNVs, as well as variants of uncertain clinical significance (VOUS); examine LOH larger than 5 Mb; and compare the characteristics of each vendor's provided visualization software.

In the background of the late onset and the progression of some mtDNA disorders there is good evidence for increases in the proportion of some pathogenic mutations - including pathogenic large scale deletions and tRNA point mutations - with age in skeletal muscle, however this is not a general phenomenon [ 2].

Nevertheless, since the highest proportion of strains pathogenic to humans are contained within clade 1, this RA approach could be an effective tool for discriminating among these strains.

*The percentage refers to the proportion of all pathogenic bacterial and fungal isolates.

Elevated viremia in untreated patients is associated with significantly higher proportions of potentially pathogenic Veillonella, Prevotella, Megasphaera and Campylobacter species than in healthy controls (Dang et al., 2012).

Approximately 100,000 species of fungi have so far been described, but only a very small proportion of these are pathogenic [1].

However, we hypothesize that in the case of the SPINK1 N34S high-risk haplotype, the variation in reported effect sizes may be a result of differences in the proportion of subjects with pathogenic pathways linking environmental stressors to pancreatic stellate cell (PSC) activation through recurrent trypsinogen activation and inadequate trypsin inhibition by SPINK1.

The number and proportion of patients with pathogenic microorganisms were slightly higher in the EcN group (27/55 patients) than in the placebo group (21/58 patients) at baseline.