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Axokine is a genetically engineered form of ciliary neurotrophic factor, a protein that promotes survival of nerve cells.
In most cases, stress promotes survival because it forces organisms to adapt to rapidly changing environmental conditions.
The onset of fever in response to invading pathogens potentiates the immune response and promotes survival.
This scenario suggests that reducing mitochondrial oxidation not only promotes survival of cancer cells but also increases anabolic metabolism.
It stimulates caspase8-dependent apoptosis, and/or promotes survival mainly through PI3K/AKT and NFκB pathways (Mocellin and Nitti, 2008; Balkwill, 2009; Waters et al., 2013).
RGD promotes survival of hMSCs encapsulated in PEG gels and has shown to induce early stages of chondrogenesis, while its persistence can limit complete differentiation.
In young healthy animals evidence suggests triggering the sterile inflammatory response promotes survival, facilitating innate immune responses to potential infection after injury.
This result shows that iota-carrageenan promotes survival of influenza A virus-infected HNep cells.
Csk1 promotes survival of DNA damage, presumably through one or more downstream CDKs.
Although STAT5 promotes survival of hematopoietic progenitors, STAT5−/− mice develop mild neutrophilia.
Exercise increases cell proliferation and promotes survival of adult-born neurons in the dentate gyrus.
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CEO of Professional Science Editing for Scientists @ prosciediting.com