On the basis of the above findings, it seems that improved nutritional management promotes post-natal ponderal growth in pre-term infants with BPD.
By comparison of WT, PrP−/− and PrP+/+ mice, we for the first time show that PrPc promotes post-ischemic neurogenesis and angiogenesis.
Bellovin et al. [ 14] reported that RhoC expression and activation are induced by EMT of colon carcinoma cell and RhoC promotes post-EMT cell migration.
The findings of this study suggest using nutritional supplements to increase enteral energy intake in association with an adequate protein intake promotes post-natal ponderal growth in pre-term infants affected by BPD.
Indeed, whereas LDC has negligible effects on primary tumour growth while improving post-surgery survival, sunitinib has marked effects on the primary tumour but promotes post-surgical metastatic dissemination.
We used the refractory period (the time between the 1st and 2nd spicule insertion) as a metric to identify the cellular source(s) that promotes post-coital mating attenuation.
HIF-1 α controls NPC migration and trafficking via PACAP-38, which is de novo expressed in the ischemic brain and itself promotes post-ischemic neurological recovery and neuronal survival.
TGF-β effects on tumors and the post-MI LV are likely opposite, as TGF-β promotes post-MI infarct healing and blocking TGF-β increases MI-induced mortality and LV dilation [ 105].
For example, chronic alcohol consumption increases the risk and severity of chronic infections with HIV; hepatitis C virus (HCV); or Mycobacterium tuberculosis, the bacterium that causes tuberculosis, and promotes post-trauma immunosuppression (for more information, see the articles in this issue by Bagby and colleagues, by Dolganiuc, by Molina and colleagues, and by Simet and Sisson).
To understand the implications of the altered lncRNAome in post-stroke pathophysiology, we currently evaluate whether ischemia promotes the association of lncRNAs with the CMPs that act as corepressors of the transcription factor REST. Recent studies showed that REST up-regulation promotes post-ischemic neuronal death (Noh et al., 2012; Pandi et al., 2013).
