Mutational hotspotting can promote evolution, including evolution of tumors and pathogens, in important ways.
Various bat species that emit high-frequency squeaks to detect prey and avoid obstacles share a high degree of variation in the FOXP2 gene, according to a new study, suggesting that genetic changes in the gene helped promote the evolution of this ability.
This could promote the evolution of mutualistic interactions by isolating less mutualistic symbiont patches from more mutualistic ones (Wilson et al. 2003; Lion and van Baalen 2008), facilitating the ability of hosts to direct resources to patches of high-quality mutualists (Bever et al. 2009).
Whether networks with higher complexity tend to adapt faster or slower in our system and whether this effect could promote the evolution of complexity remain to be tested.
This advantage would promote the evolution of anchoring proteins and other mechanisms supporting close association, and hence of biofilms and similar multicellular assemblages.
Future alteration to the pressures exerted upon the pathogen, such as climate change, environmental stress, host resistance mechanisms and human intervention, could shift this balance and promote the evolution of hypervirulence, when increased pathogen motility becomes epidemiologically beneficial.
This harmful effect is expected to promote the evolution of mechanisms that reduce the risk for females to fertilize their eggs by sperm from distorter males.
In this way, meiotic drive alleles can even promote the evolution of chromosomal rearrangements, in spite of their fitness costs.
They also promote the evolution of the law by supporting the development of legal theories that may not gain acceptance the first time around.
These attempts to overcome the original limitations include various new mechanisms proposed to promote the evolution of the Internet[2, 3].
