Sentence examples for prominent adhesion from inspiring English sources

By applying the JKR type theory for power-law graded solids to each individual asperity of the rough surface, the total normal forces for the rough surface are derived for loading and unloading stages, respectively, and a prominent adhesion hysteresis associated with dissipation energy is revealed.

Although these molecules are common to different inflammatory sites, many of the prominent adhesion proteins expressed in the inflamed rheumatoid synovium are also expressed in cartilage.

The interaction was greatly enhanced in PIP2-activated vinculin (Hüttelmaier et al., 1998) but the relevance of this interaction needs to be clarified especially since zyxin, another prominent adhesion component, has four proline-rich regions with stronger binding to VASP than vinculin (Reinhard et al., 1996).

On fibronectin or collagen I, cells formed large actin stress fibres and prominent focal adhesions, while on laminins fewer stress fibres and smaller, more-peripheral focal-adhesion-like structures (sometimes termed 'focal contacts') were observed (Refs 9, 10, 11).

The prominent peripheral adhesions that contain activated c-Src were dynamically regulated, compared with the static, more rudimentary peripheral adhesions in the parental non-metastatic cells, as judged by time-lapse imaging (not shown).

MYTP1 inhibition results in more prominent focal adhesions and absence of cell migration [ 19].

The map of the intensity derivative in the small region (64 × 64 pixels) shown in Fig. 6 contains several areas with prominent, bright adhesions (Fig. 6 G ).

Signalling through laminin-binding integrins often features minimal activation of RhoA, which controls the formation of actin stress fibres and prominent focal adhesions, and strong activation of Rac1 and Cdc42, which control the formation of lamellipodia and filopodia, respectively.

Cadherin is a prominent homotypic cell adhesion molecule that plays a crucial role in establishment of intercellular adhesion.

The most prominent embryonic integrin-adhesion structures are the muscle attachment sites (MASs); without integrin or talin, the muscles fully detach.

These findings indicate that elevated expression of active c-Src in the non-metastatic KM12C cells is sufficient to confer an enhanced ability to spread on underlying matrix components by forming prominent integrin-dependent adhesions.