Sentence examples for prolongation from from inspiring English sources

Some protease inhibitors have been shown to prolong the QT interval; however, this patient was not on a protease inhibitor and QT prolongation from other antiretrovirals has not been reported.

In the distribution computation step, due to the multilevel meshes iteration scheme, the source may be blurred due to the distribution prolongation from the coarser mesh to the finer mesh.

Production of active tyrosinase increased steadily from 0 to 13 U mL-1 after 6 h induction, and increased only slightly from 13 to 13.6 U mL-1 with the prolongation from 28 to 40 h.

As no survival prolongation from splenectomy has been demonstrated, the risks of splenectomy should be carefully balanced against the possible benefits in every individual patient, in order to restrict the procedure to those patients most likely to benefit from it.

These tests are usually based on the ECG: QTc interval prolongation from in vitro animal models (such as wedge preparations and Langendorff-perfused heart), in vivo animal models, clinical trials, and eventually human thorough QT trials, specified by the ICH E14 document.

After the completion of dose level 1, the study protocol was amended to exclude patients with congenital long QT syndrome, QTc ⩾460 ms on baseline ECG, history of QT prolongation with other medications or on medications that potentially affect QTc intervals after one death was reported in a patient with progressive QTc prolongation from another phase I study (Mahadevan et al, 2011).

QTc interval prolongations from a predose baseline of >30 ms (32 and 33 ms, respectively) were observed in 2 subjects 4 h after dihydroartemisinin-piperaquine administration.

The apparent islands of uncalcified cartilage seen in 2D sections are parts of these prolongations from the uncalcified cartilage and the viability of their constituent cells may be maintained by diffusion from the latter.

In this study, we have shown that abrogation of dNTP biosynthesis, which is caused by csn1Δ, ddb1Δ or cdc22-M45 mutatiors or HU treatment, markedly prolongs the duration of horsetail nuclear movements and that this prolongation results from activation of the Cds1-dependent DNA replication checkpoint.

Lamming et al. [ 4] have shown that decreased mTORC1 signaling is sufficient for lifespan prolongation independently from changes in glucose homeostasis.

The absence of T2 prolongation results from the dense cellularity and high nucleus-to-cytoplasm ratio of lymphoma, and it may help in the differentiation of primary CNS lymphoma from other brain tumours [ 1- 3].