"proliferation of virus" is a correct and usable phrase in written English.
You might use it to describe the rapid spread of an infectious virus, as in the following example: The proliferation of the virus caused panic in the small town.
The proliferation of virus results in immense number of deaths each year because of unavailability of on-shelf drugs.
For example, WNV cases in humans are associated with the proliferation of virus in avian amplification hosts, but not with infected equines or humans because the latter two species produce little viremia47.
Unavailability of on-shelf drugs for the prevention of further proliferation of virus inside the human body results in immense number of deaths each year.
In the early stage of antiviral development, the phenotype-based screening was launched to find reagents that can inhibit the proliferation of virus in the cell-based assays.
Early events during influenza infection dictate the outcome of the disease by containing and preventing excessive proliferation of virus in the respiratory tract, which is necessary for the adaptive immune response to mount an effective retaliation against the invading pathogen.
With the proliferation of people on the planet has come a proliferation of viruses.
The most important reason for the proliferation of viruses is the explosive growth of corporate networks, often hooked to the Internet.
The more biologically relevant response, namely the proliferation of virus-specific memory T cells, was somewhat less for the paramyxoviruses compared to IAV.
Live virus was more stimulatory than inactivated virus in inducing DC-mediated proliferation of virus-specific memory CD4+ T cells, suggesting a lack of strong suppression by live virus.
To determine the contribution of lung-resident CD103+ and/or CD11bhi RDC to the induction of an in vivo adaptive immune response in the draining MLN following influenza infection, we examined the impact of elimination of CD103+ and CD11bhi RDC from the respiratory tract on the activation and proliferation of virus-specific naïve CD8+ T cells in the DTR model.
Interactions of E6 with this set of proteins thus enhance the proliferation of virus-infected cells and cumulatively add to the oncogenic potential of HR-HPVs.
