Background: Desmoid tumors are neoplasms characterized by proliferation of fibroblasts without cytologic features of malignancy.
PGS-CinA elastomers are intrinsically cell-adherent and support rapid proliferation of fibroblasts.
The stiffer the substrates were, the stronger the abilities of adhesion and proliferation of fibroblasts were.
In the wound healing processes, the migration and proliferation of fibroblasts are essential.
A preliminary study on the proliferation of fibroblasts on the developed surfaces was performed using the NIH/3T3 cell line.
Objective: To determine the effect of hypoxia on apoptosis and proliferation of fibroblasts cultured from normal peritoneum or adhesions.
The cell fixation results from 1 and 7 days of incubation indicated the attachment, spreading and subsequent proliferation of fibroblasts.
In vitro studies involved the investigation of the proliferation of fibroblasts Rat-1 and epithelial cells IAR-2 at the tested films and morphometric analysis of the cells cultivated on the films.
Spreading and proliferation of fibroblasts are loosely governed by the substrate stiffness within the range of Young's moduli in PGS-CinA networks that were prepared.
Attachment and proliferation of fibroblasts on starPEG-heparin hydrogels of differing stiffness, density of pro-adhesive RGD peptides and MMP cleavable peptide linkers were tested.
In our experimental results, histologic findings in only electrospun SF scaffolds showed more proliferation of fibroblasts in deep layer and more differentiation of keratinocytes in superficial layer.
