Sentence examples for proliferation competence from inspiring English sources

This finding may correlate with RBR1 expression in proliferating cells, such as root meristems [54], and may function to maintain proliferation competence.

It was suggested that CDKA levels correspond to proliferation capacity of plant cells [49], and thus it appears that in AP2C3oe this proliferation competence is maintained.

Consistent with previous observations in FA cells corrected by LV-mediated gene therapy (Raya et al, 2009), this proliferation competence of FA-corrected cells was particularly remarkable when samples were subjected to cell reprogramming, confirming the relevance of the FA pathway during the process of iPSC generation.

To clearly distinguish the potential to proliferate (competence) and actual proliferation, we introduce terms competence and incompetence (permanent loss of proliferative potential associated with cellular senescence).

Such cells display uncontrolled proliferation, metastatic competence and tumorigenicity (Herlyn et al 1985 , 1987.

This complex task comprises regulation of stemness, cell proliferation, targeted migration, competence, induction, and primary formation of nephrons [9].

Furthermore, we show that transcriptional activity of the ES directly determines cell proliferation and developmental competence via a subset of expression site associated genes (ESAGs).

Disruption of critical cell-signaling pathways responsible for cell growth and survival has been shown to contribute to atypical cell proliferation, to metastatic competence of breast cancer cells, and may be responsible for therapeutic resistance of some breast cancers.

A variety of vasculopathies shift the phenotype of SMC from one of quiescence and contractile competence to proliferation, migration, matrix production, and attenuated expression of contractile proteins [1].

The generally unrestricted gene expression pattern suggests a role for progranulin in cell survival or proliferation or as a competence factor.

Evidence suggests that fetuses and children are more sensitive than adults to the toxicity of many environmental air pollutants because of their higher cell proliferation rates, lower immunologic competence, and decreased ability to detoxify carcinogens and to repair DNA damage (Anderson et al. 2000; National Research Council 1993; Perera et al. 2004; World Health Organization 1986).