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Thus, the ability of p53 to act as a defense against tumor progression was shown to be age-dependent [ 111].
Aberrant mitotic progression was shown to generate de novo DNA damage (Colin et al., 2015; Ganem and Pellman, 2012; Hayashi and Karlseder, 2013).
The predictive effect exerted by total relapses before progression on the accumulation of disability during secondary progression was shown to come exclusively from Years 1 and 2 relapses (Table 11).
Molecular mechanism of Notch-mediated cell-cycle progression was shown to involve activation of c-myc [ 36, 42], NFAT [ 43, 44], and AKT/PI3K pathway and inhibition of PTEN expression [ 45].
The mechanism of CCN6-inhibited breast cancer progression was shown to be mediated by the BMP4/TAK1/p38 pathway, which could induce epithelial-mesenchymal transition, cell invasion, and metastasis [ 88].
In Barrett's oesophagus, a reduced expression of heat shock proteins in metaplasia followed by a significant increase in expression during progression was shown (Doak et al, 2004; Ostrowski et al, 2007).
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Sites of radiological progression are shown.
Metabolic response and progression are shown in Figs. 1 and 2, respectively.
Each experiment for specific k the number of subsets for windows during the stream progression is shown with a base line.
Results of tumor progression are shown in figure 3.
Patterns of progression are shown in Table 3.
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