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In 2 patients with follow-up biopsies, no fibrosis progression was identified.
Disease progression was identified when any site bled at more than half of the recall visits over a period of 2 years.
Progression was identified using a modified pointwise linear regression (PLR) method and a modified Katz criterion.
In the absence of measurable lesions, the disease was considered stable unless a clinical progression was identified based on the judgment of the treating physician.
Similarly, a significant improvement in time to progression was identified for MGB-2 expressing patients, both in terms of mRNA and protein levels.
We used the clinical examination date that plaque incidence or progression was identified as the date of diagnosis; otherwise follow-up was censored for participants who remained stable or free of carotid plaque at the end of follow-up.
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The first global lipidomic profiles associated with urothelial cancer of the bladder (UCB) and its clinical stages associated with progression were identified.
Four main themes around disease progression were identified.
Several candidate genes identified in previous studies of ovarian cancer progression were identified in the PKA OVCAR model system by microarray and validated by RT-PCR.
While more DLB than AD participants were prescribed neuroleptics in the present study, no differences in rate of progression were identified.
Cumulative and annual incidence rates were estimated using life tables, and risk factors for progression were identified using Cox regression analysis.
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