The higher antiproliferative activity of 406-nm GEM-ANPs could be attributed to the S phase arrest during cell cycle progression (Table 2).
Increased expression of RASGRP1 and RasGEF (ARHGEF9) may contribute to Ras-ERK signaling that regulates cyclin expression and thus cell cycle progression (Table 3, network 1).
The ROC analysis demonstrated that FR, weight-adjusted FR, and plasma NGAL could significantly predict AKI progression to stage 3; in contrast, urinary L-FABP and NAG levels could not predict AKI progression (Table 2).
Several functional categories have been shown to be important for carcinogenesis and cancer progression (Table S2).
However, in the same multivariate analysis, the Gleason sum had not independent prognostic value of the biochemical progression (Table 3).
The primary component of focal adhesions, FAK, did not exhibit significant changes in mRNA levels during MOSE progression (Table 2).
Albuminuria was associated with this non-dipper progression (Table 4).
Eicosanoid compounds have been studied as targets for drug development to control diabetes progression (Table 1).
The patient treated with pazopanib had rapid disease progression (Table 2).
Next, we assessed the correlation between CTCs in blood and tumour progression (Table 1).
In this case, the carrying capacity can tremendously decelerate tumor progression (Table 3).
