Sentence examples for progression of decay from inspiring English sources

Specifically, localized radiation therapy ―as used clinically for treating cancer― given at the lesion site in transected rat spinal cord within 2 3 weeks postinjury can for the most part halt the onset and progression of decay processes; consequently, some structural and functional repair is facilitated [4] [5], [10].

Specifically, localized radiation therapy ―as used for curing cancer― given at the lesion site within a critical time window, 2 3 weeks postinjury, seems for the most part to halt the onset and progression of decay processes; consequently some repair is facilitated in transected [3], [5], [21] and in the severely contused [20] rat spinal cord.

In addition, research on the progression of wood decay in trees suggests that the origin of wood decay can be in the sapwood, rather than from saprotrophic growth in non-functioning heartwood (Boddy and Rayner 1983; Parfitt et al. 2010).

Fluoride rinses and gels, and varnishes applied by a dentist, may be able to halt the progression of root decay and in some cases reverse the damage.

In the slow progression of normal decay, chaparral litter seems to give up to the soil what have been vaguely described as "waxlike complexes of long-chain aliphatic hydrocarbons".

There is anecdotal but not scientific evidence that fluoride allows more time for dental treatment by slowing the progression of tooth decay, and that it simplifies treatment by causing most cavities to occur in pits and fissures of teeth.

Previous studies suggest that the rapid progression of Y gene decay occurred shortly after the initiation of the sex chromosome differentiation in eutherian [ 4, 14, 15].

Synaptic strengthening was determined by examining the temporal progression of the linear slope decays of fEPSPs induced after high frequency stimulation normalized to respective baseline values.

However, all these approaches have one crucial aspect in common, i.e. the need for defining a minimum threshold for wood MC that is necessary for the onset of decay and its subsequent progression.

The present study showed that Ca2+ sparks decay with progression of diabetes, accompanied by a successive impairment of cardiac function.

Ca2+ sparks exhibited a time-dependent decay with progression of diabetic cardiomyopathy, which may partly contribute to cardiac dysfunction.