Sentence examples for progression material from inspiring English sources

Stat5a and Stat5b protein levels were quantified in situ in breast-cancer progression material.

In contrast, total cellular Stat5b levels remained unchanged across the progression material.

With selective Stat5a and Stat5b antibodies and immunofluorescence detection on the Automated Quantitative Analysis (AQUA) platform [ 33, 34], Stat5a and Stat5b protein levels were quantified in breast cancer progression material.

In contrast, our initial study of a breast cancer progression material by using a pan-Stat5a/b antibody indicated that malignant breast tumors generally remained positive for expression of Stat5 protein across the progression series, although quantification was not attempted, and the antibody used did not discern between Stat5a and Stat5b [ 29].

Clinical information was not available for progression Material I. Patient demographics and clinical features of Materials III, IV, V, and VI are presented in Table 1.

This review highlighted recent progression in material models and FE simulations endorsed by experimental testing.

Perturbations in the proteolytic capacity of endosomes may not only impinge on protein turnover within but also affect progression of material along the endocytic subcompartments.

The testes of Oncopeltus show an organisation typical of insect testes (Dumser, 1980), with stages of spermatogenesis located in an anterior posterior progression (supplementary material Fig. S4).

This percentage was significantly higher than what was observed in control littermates, suggesting that Nde1−/−Lis1+/− progenitors had a normal speed in entering the M-phase from the S-phase, but delayed in M-phase progression (Supplementary Material, Fig. S3).

Given the role of Stra8 in meiosis, a greater percentage of the Stra8-GFP+ cells had initiated meiosis (up to 10 15%), but these cells also exhibited a block in meiotic progression (Supplementary Material, Fig. S5D).

To determine whether Xic1's role in differentiation is solely to promote cell-cycle exit in cardiomyocytes, we rescued Xic1Mo-induced loss of cardiac differentiation with different Xic1 deletion constructs, which all have the ability to inhibit cell-cycle progression (Supplementary material online, Figure S3A ) but by different mechanisms.