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Previous theoretical studies of the evolution of incompatibility-inducing systems and their dynamics have found selection among parasites and hosts to favor variants that increase the proportion of infected progeny, i.e. by increasing transmission rates [21].
Here, we investigated the fate of these IhNSC's immediate progeny (i.e. neural progenitors; IhNSC-Ps) upon unilateral implantation into the corpus callosum or the hippocampal fissure of adult rat brain, 3 days after global ischemic injury.
For each site, the mating success of females and proportion of females that produced live progeny (i.e., those females whose eggs overwintered successfully to hatch) were estimated as described in table 1. Sampling was conducted in Indiana (Fig. 2) throughout the emergence period of adults in late summer and early fall (26 sites for arborvitae and 24 sites for juniper).
Note that the number of time points measured may be progeny-specific, expressed as m ifor progeny i.
The photosynthetic rate for each progeny i (i = 1,..., n) is measured at different irradiance (s = 1,..., S) and temperature (t = 1,..., T ) levels.
Let denote the vector of phenotypic values for trait k (k = 1 for leaf biomass (L), 2 for stem biomass (S), and 3 for root biomass (R)) measured on progeny i at time points.
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Based on the available pedigree, it is possible to apply Mendelian rules to estimate the percentage of genotype conflicts for all SNPs in father-progeny pairs (i.e. progeny's genotype is alternatively homozygous to father's homozygous genotype) for an imputed progeny compared to each sequenced individual.
For both transgenes, T1 progeny displayed (i) resistance to PVA infection, (ii) susceptibility, or (iii) systemic infection followed by recovery of new leaves from PVA infection (RC), regardless of the transgene.
Positions that met all of these criteria were considered candidate positions for progeny genotyping, i.e. the Dd2 base call differs from the HB3 base call.
For our purpose, this approximation leads to an apparent contradiction, because it implicitly assumes no drifts in allele frequencies between parent and progeny generations (i.e. Neb = ∞).
Simultaneous estimation of female and male recombination rates from intercross data in outbred organisms is an easy task if the parental gametes can be deduced from the progeny genotypes, i.e., when at each of the two marker loci 3−4 alleles are observed in the progeny.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com