Sentence examples for products age from inspiring English sources
ROS are generated on one hand by stimulation of the glucose metabolism and the respiratory chain in the mitochondria and on the other hand by the production of advanced glycation end products (AGE).
Hyperglycemia leads to modifications of proteins by nonenzymatic glycation as described elsewhere [ 4, 44], forming advanced glycation end products (AGE), which are sources of free radical production according to Liguori et al. [ 44].
"On a like for like basis, against properly comprehensive products, Age UK Funeral Plans are in fact very competitively priced".
The interaction between advanced glycation end products (AGE) and their receptor RAGE mediates the progressive alteration in renal architecture and loss of renal function in diabetic nephropathy.
This induces the irreversible formation of advanced glycation end products (AGE), which may alter protein function and protein degradation.
Advanced glycation end products (AGE) are substances that can induce insulin resistance in adipocyte, hepatocyte and muscle cells.
The study of Tamami et al. [ 20] demonstrated the inhibitors of the transcription factors NF-κB completely prevented the advanced glycation end products (AGE -induced up-regulAGE -inducedF mRNAs and the subsequent increase in DNA synthesis in endothelial cells.
17 Hunt et al indicate that high levels of oxLDL-IC and advanced glycation end products (AGE -LDL-IC AGE -LDL-ICnt predictors of cAGE -LDL-ICmareedimportantening in predictorsith type 1 diabetes.
Advanced glycation end-products (AGE) and the receptor for AGE (RAGE) have been linked to numerous diabetic vascular complications.
Knowing that use of PD fluid containing GDPs generates advanced glycation end-products (AGE) [ 80], a comparison of wild-type mice with mice deficient in AGE receptor (RAGE) was performed.
The present study was designed to investigate whether the effects of dietary supplementation with α-lipoic acid could prevent the increase in mitochondrial superoxide production in the heart as well as the enhanced formation of advanced glycation end-products (AGE) that are associated with the development of hypertension and insulin resistance in chronically glucose-fed rats.
