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In other bacteria swarming involves differentiation into an elongated cell type, hyperflagellation and the production of slime.
The production of slime and the ability to form biofilm has therefore been reported to play the most important roles in the pathogenesis of S. epidermidis and other CoNS-induced infections [ 5].
The production of slime, lipase, lecithinase, nuclease (DNAse), thermonuclease (TNAse), α- and β-hemolysin, enterotoxins (SEA, SEB, SEC, SED) and toxic shock syndrome toxin-1 (TSST-1) was studied in S. aureus and CoNS.
The objective of the present study was to compare the capacity of traditional clinical and bacteriologic and selected virulence factors such as production of slime, enzymes and toxins to predict the outcome of new peritonitis episodes caused by S. aureus, S. epidermidis and other CoNS.
Finally, by a simple visual observation of the colony advance edge of both the wild-type and Δ recA strains, we can rule out any effect of RecA on the production of slime because the slime that normally surrounds the colony periphery was, apparently, similar in both cases (data not shown).
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Despite the small number of agr III isolates, milk had a positive effect (P < 0.05) on biofilm production regardless of slime production.
Another proposed mechanism included the increased production of a 'protective slime' composed of precipitated PG complexed to fatty acids [25].
In this study, the production of biofilm and slime by S. aureus isolated from mastitic cows was investigated.
Salicylic acid inhibited the production of teichoic acid, slime-associated proteins, and polysaccharide/adhesin production by S. epidermidis [17].
Tests for the production of haemolysin, protease, lipase, lecithinase, slime, leucocidin toxin were performed.
Biofilm formation is related to the production of an extracellular mucoid polysaccharide, called slime, which permits microorganisms to adhere to plastic surfaces [ 12].
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