Finally, one of the biggest challenges in the understanding of the aging process is to decipher the connections between the seemingly independent molecular events that have been reported in different settings of senescence.
This information also helps to decipher processes involved in illness, for instance the molecular heterogeneity of cancer [ 5].
Since the majority of patients in the PVL, DWMG, and Negative groups required mechanical ventilation of comparable durations, and showed substantial involvement by inflammatory/infectious processes, it is difficult to decipher the factors responsible for the substantial brain injury in the PVL group.
Overall representativeness is not a trivial outcome, as multiple artifacts may bias the overall result during the process, making it impossible to decipher the causes of distortions.
To help more environmental scientists use voltammetry to decipher microbial processes that mediate redox conditions in undisturbed soils and sediments, this paper shows how to quantify the voltammetric signals of O2, Mn II), Fe(II), and S −II) and overcome challenges imposed by the use of solid-state electrodes.
However, long-term analyses, over time-spans of centuries, are needed to decipher: the processes controlling the occurrence of disturbances; their affects on ecosystem properties; and, feedbacks to disturbance regimes [3], [4].
Of equal importance, these studies suggest that methyl sulfone may be a tool for basic scientists to decipher fundamental processes whose mechanisms are not well understood: what induces or reverses contact inhibition; what regulates induction of senescence; what signaling pathways play a role in the transformation of healthy cells to cancer cells and back to healthy cells.
These results open up new lines to decipher the processes controlling grape berry development and ripening.
The fact that a single amino acid substitution of arginine for histidine in the FHA domain of Unc-104 is sufficient to perturb all these processes presents an ideal tool to decipher the biophysical underpinnings of neurodevelopmental disorders.
Current transgenic or knockout mouse models might indeed provide insights into the early stages of PD (Fleming et al. 2005), allowing us to decipher pathological processes that take place in brain exposed to a PD-causing insult before degeneration of the nigrostriatal system occurs, or before the disease begins elsewhere (Litvan et al. 2007).
