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However, the dissociation of portal hypertension and SC imaging as seen in our study is not entirely surprising as a prior study demonstrated that the degree of abnormality detected on SC SPECT scans was not necessarily directly due to portal hypertension but related to impaired SC extraction by the reticuloendothelial cells of the liver [47].
A prior study demonstrated that injection of PV rectally into mice results in significant keratin tyrosine phosphorylation in colon tissue, as determined by using anti-pan pY antibody (22).
A prior study demonstrated that protein Rib [ 7] was present in more invasive versus colonizing serotype III strains.
This prior study demonstrated that 815 genes (19.78 % of the genome) underwent changes in expression during a four-day cyst development period.
A prior study demonstrated that the number of circulating hemocytes declines with age in mated females (Mackenzie et al., 2011), but did not analyze the sessile cells.
Our prior study demonstrated that MRglc measured with SKM had a very high correlation with that measured by kinetic modeling [ 15].
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It also agrees with prior studies demonstrating that little antigenic change occurs within a single influenza season and that genetic diversity within seasons is stochastic via chance introduction of divergent isolates [19].
The finding that amyloid positive individuals show selective thinning within the medial orbital frontal cortex is in agreement with one prior study demonstrating that older individuals with subjective memory complaints show significant correlations between gray matter atrophy and neocortical PiB increase within the medial orbital frontal/anterior cingulate cortex [52].
Our prior studies demonstrated that the majority of cortical SR is expressed in glutamatergic neurons.
Our prior studies demonstrated that exogenous supplements of pure hyaluronan (HA) tetramers (HA4) dramatically upregulate elastin matrix synthesis by adult vascular smooth muscle cells (SMCs).
Because prior studies demonstrated that FLCs generated intracellular oxidative stress, our studies focused on the redox-sensitive mitogen-activated protein kinase kinase kinase known as apoptosis signal-regulating kinase 1 (ASK1).
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