(B) Primary skin fibroblast cells from the HBB −28 (A>G) mutant patient.
All of these pathogens can cause primary skin and soft tissue infection [8, 31, 32].
Then we collected primary skin fibroblast cells from a β-thalassemia patient with HBB −28 (A>G) homozygous mutation.
There is a strong epidemiological link between CA-MRSA causing deep primary skin infections and Panton-Valentine leukocidin (PVL).
Its epicentre in the cutaneous layer, irregular and infiltrative appearance should raise suspicion for a primary skin malignancy.
We presented two cases where acute deformation of the fractured tibia allowed primary skin closure and avoided the need for soft tissue flaps.
Intriguingly, we found that HBB −28 (A>G) mutation repairing efficiency was about 20% in the constructed cell line and primary skin fibroblast cells.
Open image in new window Figure 2 Correcting HBB −28 (A>G) mutation in primary skin fibroblast cells of beta thalassemia patient.
The photoprotective potential of this compound against UVA-induced oxidative damage and cell death was evaluated in cultured primary skin fibroblasts.
To evaluate the role of Wnt-10b in epithelial differentiation, we investigated the effects of Wnt-10b on adult mouse-derived primary skin epithelial cells (MPSEC).
In the present study we have explored mycolactone effects on human primary skin keratinocytes in vitro.
