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Our previous data indicated that a Myxococcus xanthus sensor-type adenylyl cyclase (CyaA) functions in signal transduction during osmotic stress.
The previous data indicated that the most suitable carbon source, nitrogen source, salt and vitamin were glucose, yeast extract, KH2PO4 and Ca D-Pantothenate, respectively.
Previous data indicated that cells overexpressing TIP5 grow more slowly than parental cell lines [17].
All the previous data indicated that BTV-4 caused a lethal infection in adult IFNAR mice.
Our previous data indicated that DT-GMCSF induced cell death was dependent on the adaptor protein FADD [10].
Previous data indicated that βTrCP uses the non-canonical UbE motif for GHR endocytosis, although the canonical DSG motif is present in the GHR cytosolic tail.
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These findings clearly corroborate previous data indicating that these periodontopathogenic bacteria may not necessarily be related to peri-implantitis [6].
This is in line with previous data, indicating that anti-angiogenic treatments indeed significantly reduce the uptake of targeted therapies in patients and in several animal models [32 36].
While we cannot exclude this may have influenced our result, previous data indicate that flow rate does not influence plasma volume expansion by crystalloids [13].
These results complement closely with previous data indicating that (a) rac-BHFF suppressed operant, oral alcohol self-administration in sP rats and (b) the prototypic GABAB PAMs, CGP7930 and GS39783, reduced alcohol drinking in sP rats.
In summary, it would be tempting to say that ours and the previous data indicate that MSCs might be regarded as personalized modular therapies limiting short- and long-term acute lung injury severity by fine-tuning inflammation and tissue remodeling.
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