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Anticoagulation with heparin was required to prevent left ventricle thrombus formation [18, 24, 27].
An intra-aortic balloon pump (IABP) was inserted and used to keep systolic blood pressure low and prevent left ventricular rupture.
Simulated increases in afterload augmented sunitinib cardiotoxicity in both rat and human microtissues, which suggest that antihypertensive therapy may be a strategy to prevent left ventricular dysfunction in patients treated with sunitinib.
Anticoagulation with heparin was required to prevent left ventricle thrombus formation [ 18, 24, 27].
Therefore, the purpose of this study was to investigate whether a DPP4 inhibitor, vildagliptin, can prevent left ventricular (LV) hypertrophy and LV diastolic function in isoproterenol-treated rats.
By two-dimensional echocardiography and postmortem morphometry, transplanted EPO-engineered fibroblasts did not prevent left ventricular (LV) dysfunction and adverse LV remodeling 5 and 9 weeks after MI.
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