Sentence examples for presumed interaction from inspiring English sources
The phrase "presumed interaction" is correct and can be used in written English
It refers to an interaction that is assumed or believed to have taken place, even if there is no concrete evidence or confirmation. An example of using this phrase in a sentence could be: "The study's findings suggest a strong correlation between the increase in online classes and a decrease in presumed interaction among students."
However, in the same transcriptional assay AmphiVent1 was not able to influence the Lef/Tcf-responsive reporter gene (Fig. S4) despite high sequence conservation of AmhiVent1, Xvent-2 and VENTX homeodomains (Fig. 1A), which are the presumed interaction domains.
The method by which Mus81 recognizes its substrates is not well understood, but it seems possible that Anc1, through its presumed interaction with histones [13], allows the Mus81 endonuclease access to sites where its cleavage will initiate the sister-strand recombination that drives error-free PRR.
Future studies in T. belangeri will address presumed interactions of neural crest cells and placodal cells during the development of the ciliary ganglion and its associated structures.
Some of these cases arose from presumed interactions with zoonotic reservoirs or hosts, such as primates and other mammals during hunting trips (Boumandouki et al., 2005; Nkoghe et al., 2005, 2011; WHO, 2003) or butchering of bats (Leroy et al., 2009).
The presumed direct interaction between HDAC3 and PML [40] is increased slightly by atRA treatment, but this is ERK-independent.
The presumed average interaction strength is indicated in red.
The number of mitochondrial haplotypes (H mt) was increased at sites with presumed biotic interaction (bio) and to a lesser extent in the Holocene expansion sites (hol).
In our results, the patients with higher VAI values had better nutritional status (Table 1), and therefore we presumed an interaction between the VAI and nutritional status when assessing the predictive power for all-cause mortality.
One explanation for the difference in the potency observed in the cell-based assay systems is the presumed different interaction between JH1 inhibitor complex and the JH2 domain, where the mutation occurs.
Because extensive studies of the functional consequences of presumed direct interaction of Src with Na+/K+-ATPase have been reported, it was important to explore the functional status of the endogenous Src of the two kidney pools of the Na+/K+-ATPase.
Compared to the analysis of variance, the advantage of this QTL analysis is to set the location for only one of the two QTL presumed in interaction, increasing the number of birds analyzed (40 versus 29 animals) and then allowing to better localize the second QTL.
