Sentence examples for preserving beta cell from inspiring English sources

16 The findings suggest that a combination of a GLP-1 receptor agonist and insulin glargine might represent an effective strategy for preserving beta cell mass in patients with type 2 diabetes and potentially could alter disease progression.

As such, early identification of T2D, particularly in high-risk individuals, may be justified, along with earlier initiation of pharmacotherapy aimed at preserving beta cell function and minimizing long-term microvascular complications.

These early findings are now being evaluated in clinical trials to determine whether the combination of a GLP-1 receptor agonist, such as lixisenatide, and insulin glargine might represent an effective strategy for preserving beta cell mass in patients with type 2 diabetes.

The reasons for this are many, and include issues such as poor adherence to complex medication regimes; costs of prescribed therapies; and the failure of traditionally prescribed medications to preserve beta cell function over time.

Animal studies also suggest that dipeptidyl peptidase-4 inhibitor treatment may help to preserve beta cell mass; however, it is unclear at present whether or not this will prove to be the case in humans.

Most current T2DM therapeutics do not prevent continued loss of insulin secretion capacity, and those that do have the potential to preserve beta cell mass and function are not effective in all patients.

Here are two: seeing via a closed-loop system whether Symlin reduces post-meal glucose spikes, and seeing if a closed-loop system used by newly-diagnosed Type 1 kids preserves beta cell function.

Atorvastatin treatment did not significantly preserve beta cell function although there may have been a slower decline of beta-cell function which merits further study.

Participants Non-obese patients (BMI ≤27) with preserved beta cell function.

Thus, compared with GLP-1, low-dose GLP-1 oestrogen revealed superior efficacy to preserve beta cell integrity and function under diabetogenic conditions.

The increase in IL-1Ra could be an attempt by the body to counteract the deleterious effects of IL-1β and to preserve beta cell survival, insulin secretion and insulin sensitivity.