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Materials: forty-four patients, with 44 plaques associated with various presentations, were studied.
The course we chose also covered topics with which our process experts were quite familiar; results may have been different if less common clinical presentations were studied or process experts without generalist training were selected.
The clinical presentations were studied in 50 out of 148 familial AD (FAD) patients (age at onset = 4 0.0±4.8; 38% women, 18 alive) and in 9 out of 34 patients (age at onset 65.1±17.1) belonging to the FTD family.
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Results: Five hundred fifty-nine second twins and 18,061 vaginally delivered singletons in the cephalic presentation were studied.
Three patients, 53 60 years old at initial presentation, were studied.
The small number of students who were studied at the two time points and the fact that only a single presentation was studied also limits generalizability of these results to other presentations.
Such data were summarized in tables for each of the leishmaniasis forms of presentation that were studied.
Five genes involved in the presentation antigen class I pathway were studied by qRT-PCR: SLA Ia, TAP1, TAP2, PSMB8 and PSMB9.
Age at presentation, associated anomalies, and operation outcomes were studied.
The classical and effective pathway of antigen cross-presentation has been studied in detail [14].
The mechanisms of cross-presentation have been studied intensively [15] [17] while the details have been left unclear.
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